perjantai 24. lokakuuta 2014

Can natural protocols be an effective treatment for Ebola?

 October 19, 2014 

Since writing “Surprising solution for Ebola virus” and having it published by NaturalHealth365 August 3, 2014, a lot has transpired. 

The Ebola epidemic was only beginning, and it had only been featured in the nightly news for about 2 weeks at that time. Nobody, I believe, had any clear handle on what was going to happen or how the epidemic was going to evolve.

Over the 10 weeks since that article first appeared, and especially over the last 4 weeks or so, I have received a literal avalanche of emails from around the world.

Most of them were very supportive of my efforts to let those “in charge” know there were real options available to stop Ebola beyond vaccines, isolation, antipyretics, nutrition, and vigorous hydration (“feed ‘em and fan ‘em, as we used to say when I was an internal medicine resident at Tulane University in New Orleans). 

A few, however, have tried to demonize me by saying that I should be ashamed of myself for even suggesting something as outlandish as vigorous vitamin C supplementation to help facilitate recovery from an Ebola infection. One individual even suggested an article like mine should be illegal, as it threatened lives. Imagine, vitamin C threatening someone’s life! But the beat goes on…

For the record, let’s be perfectly clear about vitamin C and Ebola

One thing I would like to immediately clarify. Vitamin C has never cured Ebola virus, or to my knowledge, accelerated its resolution. Also, to my knowledge, no attempt has even been made to utilize this phenomenal vitamin/nutrient in such a fashion.
What vitamin C has done, and this has been reported in the medical literature, is to disintegrate or otherwise inactivate every virus against which it has been tested, and many different viruses have been subjected to such experiments.
As reported in my earlier article, Dr. Frederick Klenner resolved viral syndromes routinely and with dispatch by using multigram doses of vitamin C intravenously (50 grams at a time or more).
He even reported on several patients comatose with viral encephalitis. 

These individuals regained consciousness within a few hours of the vitamin C infusions and ultimately recovered, typically in days, not weeks or months.
Now, with specific regard to Ebola, it is just as important to support the body as to make it difficult for the virus to survive. Although several cases have now occurred in American healthcare workers, the rapid spread is mainly occurring in Africans that don’t have anything close to the average nutritional status of even a teenager in the United States subsisting predominantly on Chicken McNuggets.  This makes an enormous difference in the effectiveness of any therapy, including vitamin C.

When do infectious diseases cause the most harm to human health?

When Dr. Klenner treated an acute viral syndrome with intravenous vitamin C, there was typically an immune system with adequate capacity to “mop-up” the toxic debris resulting from massive viral disintegration.

However, in the typical Ebola patient in Africa, vitamin C, ozone, ultraviolet blood irradiation, and all of the other treatments known to stop viruses in their tracks will be releasing toxic byproducts into a body that has already been chronically decimated by malnutrition.
Even the best-fed individuals in the areas affected do not have a wide variety of good foods, and significant nutritional deficiencies can be expected to be present among them as well. Remember that you do not have to be pathologically skinny to actually be severely malnourished. Even obese individuals have managed to get scurvy in the past.
Simply put, nutritionally-deficient individuals run the greatest risk of premature death from infectious diseases.

How to reduce the risk of infectious diseases 

While I don’t intend to present a comprehensive potential treatment for Ebola in this article, I emphasize to anyone wanting to treat Ebola with anything to include the following considerations (I am avoiding specific dosages, since this can range very widely depending upon the clinical circumstances):

1. The ‘multi-C protocol‘ (Vitamin C, highly-dosed in multiple forms), as outlined in my previous writings on NaturalHealth365. 

2. Adequately dosed vitamins K and D – the blood level of vitamin D should end up between 60 and 80 ng/cc; proper D levels are enormously important in helping vitamin C to bolster the immune system and prevent and/or resolve an infection.
3. Adequate B vitamins.
4. Vigorous hydration, orally and intravenously.
5. High quality nutrition – as liquids and solids as tolerated.

The regular administration of vitamin C powder in such liquids can help heal the sick and malabsorbing gut present in these clinical situations; even in those with illness-induced diarrhea large amounts of vitamin C powder and nutrient-rich liquids can resolve this more quickly and allow the quality nutrients being swallowed to end up being better absorbed and assimilated.
6. Other quality supplements/nutrients. It is important to realize that many people adversely affect their digestion when very large amounts of different supplements are taken, and this should be even more significant for the chronically malnourished patient. Other supplements can certainly be taken, but not to the detriment of ingesting adequate amounts of good nutrients, liquid and solid.

Why is vitamin C so effective in killing viruses? 
A primary way in which vitamin C destroys viruses, or sets them up for destruction by the immune system, is by activating the ‘Fenton reaction’. In a nutshell, this reaction can proceed inside the virus, inside cells in which viruses are replicating, and on the surfaces of the viruses themselves. The result of this reaction that is stimulated by the presence of vitamin C, one or more transition metal cations, and the local presence of peroxide is the immediate production of hydroxyl radicals. These radicals are the most reactive oxidizing agents ever identified. As such, they radically upregulate oxidative stress and end up destroying whatever is in their immediate environment. - See more at:

Making the hospital setting a safer place for everyone

An additional important point is that it is very critical for much the same regimen to be utilized in the healthcare workers who are tending to Ebola patients, regardless of how much protective clothing is worn or antiseptic measures are being taken. The virus can only propagate once it takes hold, and it can only take hold when a large exposure is suddenly encountered and assimilated.

There is nothing a virus “wants to encounter” less than a high antioxidant presence in the blood and extracellular fluids after it gets into the body. With our current state of knowledge on Ebola, healthcare workers have every right to be anxious over their possibility of getting infected, and until more practical clinical knowledge is acquired, they need to receive pretty much everything the patient receives, as itemized above, and including intravenous administration of vitamin C.
About the author: Thomas E. Levy, MD, JD is a board-certified internist and cardiologist. He is also bar-certified for the practice of law. He has written extensively on the importance of eliminating toxins while bolstering antioxidant defenses in the body, with particular focus on vitamin C.
His new book entitled Death by Calcium: Proof of the toxic effects of dairy and calcium supplements is now available at or In this new book, for the first time, Dr. Levy has assembled extensive sections on his treatment protocols for cancer, heart disease, osteoporosis, and other chronic degenerative diseases. As well, this new book contains his detailed Guide to the Optimal Administration of Vitamin C.”
His website is

1. Baxter A (2000) Symptomless infection with Ebola virus. Lancet 355:2178-2179. PMID: 10881884
2. Levy T (2002) Curing the Incurable. Vitamin C, Infectious Diseases, and Toxins. Henderson, NV: MedFox Publishing
3. Klenner F (1971) Observations of the dose and administration of ascorbic acid when employed beyond the range of a vitamin in human pathology. Journal of Applied Nutrition 23:61-88
4. Levy T (2012) Treating influenza with vitamin C: results and mechanisms. Townsend Letter December
5. Klenner F (1948) Virus pneumonia and its treatment with vitamin C. Southern Medicine & Surgery February, 110:36-38, 46
6. Cathcart R. (1984) Vitamin C in the treatment of acquired immune deficiency syndrome. (AIDS). Medical Hypotheses 14:423-433. PMID: 6238227
7. Levy T (2011) Primal Panacea Henderson, NV: MedFox Publishing

See more at:


Cal-Mag, Stress Formula, 100 Tablets

NOW Foods Calcium and Magnesium Stress Formula with B-Compelx and Vitamin C - 100 Tablets 

Now Foods Calcium and Magnesium are essential minerals that work synergistically with one another to promote enhanced absorption in NOW Foods Calcium & Magnesium Stress Formula. NOW Foods Calcium-Magnesium Stress Formula contains calcium that is necessary for strong bones and teeth, and Magnesium is important for healthy enzymatic activityIn addition, NOW Foods Calcium-Magnesium Stress Formula provides a full spectrum of B-vitamins and Vitamin C.

Supplement Facts 
Serving Size: 2 Tablets 
Servings Per Container: 50 
Amount Per Serving / % Daily Value

Vitamin C (as Ascorbic Acid) 120 mg / 200% 
Thiamine (Vitamin B-1) (from Thiamine HCl) 10 mg / 667% 
Riboflavin (Vitamin B-2) 5 mg / 294% 
Niacin (Vitamin B-3) (as Niacinamide) 40 mg / 200% 
Vitamin B-6 (from Pyridoxine HCl) 5 mg / 250% 
Folate (Folic Acid) 800 mcg / 200% 
Vitamin B-12 (as Cyanocobalamin) 20 mcg / 333% 
Biotin 300 mcg / 100%
Pantothenic Acid (Vitamin B-5) (from Calcium Pantothenate) 20 mg / 200%
Calcium (from 80% Calcium Carbonate, 10% Calcium Citrate, 10% Amino Acid Chelate) 1.0 g (1,000 mg) / 100% 

Magnesium (from 80% Magnesium Oxide, 10% Magnesium Citrate, 10% Amino Acid Chelate) 500 mg /     125%
Choline (as Choline Bitartrate) 20 mg † Inositol 20 mg † PABA 10 mg † 

Daily Value not established. 

Fenton reaction

The Fenton reaction has importance in biology because it involves the creation of free radicals by chemicals that are present in vivo. Transition-metal ions such as iron and copper donate or accept free electrons via intracellular reactions and help in creating free radicals. Most intracellular iron is inferric (+3 ion) form and must be reduced to the ferrous (+2) form to take part in Fenton reaction. Since superoxide ions and transition metals act in a synergistic manner in the creation of free radical damage, iron supplementation must not be done in patients with any active infections or in general any diseases.'s_reagent

Oxidative stress

Oxidative stress reflects an imbalance between the systemic manifestation of reactive oxygen species and a biological system's ability to readily detoxify the reactive intermediates or to repair the resulting damage. Disturbances in the normal redox state of cells can cause toxic effects through the production of peroxides and free radicals that damage all components of the cell, including proteinslipids, and DNA. Further, some reactive oxidative species act as cellular messengers in redox signaling. Thus, oxidative stress can cause disruptions in normal mechanisms of cellular signaling.
In humans, oxidative stress is thought to be involved in the development of cancer,[1] Parkinson's diseaseAlzheimer's disease,[2][3] atherosclerosis,heart failure,[4] myocardial infarction,[5][6] fragile X syndrome,[7] Sickle Cell Disease,[8] lichen planus,[9] vitiligo,[10] autism,[11] infection,[12] and chronic fatigue syndrome.[13] However, reactive oxygen species can be beneficial, as they are used by the immune system as a way to attack and kill pathogens.[14] Short-term oxidative stress may also be important in prevention of aging by induction of a process named mitohormesi
Chemically, oxidative stress is associated with increased production of oxidizing species or a significant decrease in the effectiveness of antioxidant defenses, such as glutathione.[16] The effects of oxidative stress depend upon the size of these changes, with a cell being able to overcome small perturbations and regain its original state. However, more severe oxidative stress can cause cell death and even moderate oxidation can triggerapoptosis, while more intense stresses may cause necrosis.

Production of reactive oxygen species is a particularly destructive aspect of oxidative* stress. Such species include free radicals and peroxides. Some of the less reactive of these species (such as superoxide) can be converted by oxidoreduction reactions with transition metals or other redox cycling compounds (including quinones) into more aggressive radical species that can cause extensive cellular damage.[18] Most long term effects are caused by damage to DNA.[19] DNA damage can be induced by ionizing radiation is similar to oxidative stress, and these lesions have been implicated in aging and cancer.




Annu Rev Immunol. Author manuscript; available in PMC Nov 23, 2007.
Published in final edited form as:Annu Rev Immunol. 2005; 23: 197–223.
PMCID: PMC2092448

How Neutrophils Kill Microbes 

Anthony W. Segal



Neutrophils are highly motile phagocytic cells that constitute the first line of defense of the innate immune system.
They were first discovered by Elie Metchnikoff when he inserted rose thorns into starfish larvae and found that wandering mesodermal cells accumulated at the puncture site.
He showed these cells to be phagocytic and described the larger cells as macrophagocytes, or macrophages, and the smaller as microphagocytes, now known as granulocytes, of which by far the most numerous are the neutrophils.
The ability of these cells to engulf and degrade bacteria was logically assumed to indicate a killing function. A microbicidal function was ascribed to the contents of their abundant cytoplasmic granules that were discharged into the phagocytic vacuole containing the microbe () (Figure 1). Attention was then directed toward the characterization of the granules by electron microscopy, fractionation, and biochemical analysis. Several of the purified granule proteins were shown to kill microbes.
Figure 1
Transmission electron micrograph of a human neutrophil. Inset is an image taken from a neutrophil 20 s after the phagocytosis of latex particles opsonized with IgG (V, vacuole). The section was stained for myeloperoxidase (MPO) to reveal the electron-dense


The NADPH oxidase plays a pivotal role in microbial killing because its dys-function causes CGD, characterized by a profound predisposition to bacterial and fungal infection (), and killing is compromised under anaerobic conditions ().
Detailed reviews of the biochemistry and bioenergetics of this system have recently been undertaken (), to which I refer readers. A schematic representation of the oxidase is shown in Figure 2.
Figure 2
Schematic representation of the NADPH oxidase. Flavocytochrome b558 is a heterodimer of gp91phox, which contains the haem- and flavin-binding sites, and p22phox. Electron transport is activated by phosphorylation and translocation to the vacuolar membrane ...

Ei kommentteja:

Lähetä kommentti

You are welcome to show your opinion here!