tiistai 23. syyskuuta 2014

Diabetes

Diabetes on joukko erilaisia sairauksia, joille on yhteistä se, että veren sokeripitoisuus kasvaa liian suureksi. Avaintekijänä on insuliinihormoni: sen erityksen loppuminen, määrän riittämättömyys tai vaikutuksen heikkeneminen eli insuliiniresistenssi. 

Diabeteksen kaksi päämuotoa ovat tyypin 1 (nuoruustyypin) diabetes ja tyypin 2 (aikuistyypin) diabetes.
Tyypin 1 diabetes
Tyypin 1 (nuoruustyypin) diabeetikoita on Suomessa noin 50.000.

Syy
Elintoiminnoille välttämätöntä insuliinia ei erity, sillä insuliinia tuottavat haiman saarekesolut ovat tuhoutuneet.

Oireet
Tavallisia oireita ovat lisääntynyt virtsaneritys, jano, laihtuminen ja väsymys. Ne kehittyvät yleensä nopeasti, päivien tai viikkojen kuluessa.

Hoito
Jatkuva, elinikäinen insuliinihoito (pistoksina tai pumpulla) on elämän edellytys. Verensokerin omaseuranta ja hiilihydraattien laskeminen on tärkeää, koska insuliiniannokset sovitetaan hiilihydraattimäärien mukaisiksi. Liikunta ja terveellinen syöminen auttavat hoitamaan veriesuonien terveyttä ja ehkäisevät siten lisäsairauksia.

Hoidon päämääränä on hyvä elämä. Tavoitteena on pitää veren sokeripitoisuus sopivana. Tällöin sekä lisäsairauksien että liian matalien verensokeritasojen vaara on mahdollisimman pieni.

Sairastumisikä
Yleensä alle 40-vuotiaana. Suomessa lasten diabetes on yleisempi kuin missään muualla maailmassa.

Ehkäisy
Ehkäisykeinoja ei vielä tunneta, mutta mahdollisuuksia tutkitaan jatkuvasti.

Periytyvyys
2–5 %, jos äidillä on tyypin 1 diabetes. 6–8 %, jos isällä on. Vastasairastuneista diabeetikkolapsista vain noin 10 prosentilla on lähisuvussaan joku diabetesta sairastava.

Ensiapu
Jos insuliinihoitoinen diabeetikko tulee yllättäen huonovointiseksi tai menettää tajuntansa, kyseessä on melko varmasti liian alhaisesta verensokerista johtuva hypoglykemia, insuliinisokki. Sokin ensiapuohjeet löydät täältä.

Tyypin 2 diabetes
Tyypin 2 (aikuistyypin) diabeetikoita on Suomessa tiedossa noin 250.000. Lisäksi noin 200.000 sairastaa diabetesta tietämättään.

Syy
Haima tuottaa insuliinia, mutta se vaikuttaa heikosti tai insuliinia ei ole tarpeeseen nähden riittävästi. Insuliinituotanto voi vuosien kuluessa ehtyä kokonaan.

Oireet
Usein oireeton, kehittyy hitaasti jopa vuosien kuluessa. Todetaan usein sattumalta tai valtimosairauden puhjettua. Oireita voivat olla esimerkiksi väsymys erityisesti aterioiden jälkeen, jalkasäryt, lihaskouristelut sekä uupumus, johon uni ei auta.

Toteaminen
Katso verensokerin raja-arvot täältä.

Hoito
Painonhallinta liikunnan ja oikean ruokavalion avulla.
Tarvittaessa myös lääkehoito. Verenpaineen ja veren rasvojen hoito sekä veren liiallisen hyytymisen esto ovat tärkeitä lisäsairauksien estämseksi ja hoitamiseksi.
http://www.diabetes.fi/files/308/Ruokavaliosuositus.pdf

 
Sairastumisikä
Yleensä yli 40-vuotiaana. Lisääntyy nopeasti kaikkialla maailmassa ja yhä nuoremmissa ikäryhmissä.

Ehkäisy
Liikunta, terveellinen ruoka, painonhallinta ja tupakoimattomuus.

Periytyvyys
Riski sairastua tyypin 2 diabetekseen on 40 %, jos toisella vanhemmista on tyypin 2 diabetes. Riski on 70 %, jos molemmilla vanhemmilla on.


Maailman diabetespäivän 2012 luentoja

Diabetestutkimussäätiö järjesti Maailman diabetespäivän luentotilaisuuden Helsingissä keskiviikkona 14.11.2012. Tilaisuudessa pidettyjen luentojen aineistoja on ohessa.




Miksi Suomessa sairastutaan
tyypin 1 diabetekseen useammin kuin missään muualla maailmassa,

professori Mikael Knip:


http://www.diabetestutkimus.fi/files/94/Mikael_Knip_14.11.2012.pdf
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http://www.diabetes.fi/
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Diabeteksessa veren sokeripitoisuus kasvaa liian suureksi.

Ravinnossa olevat hiilihydraatit hajoavat glukoosiksi (rypälesokeri) ja kulkeutuvat vereen. Normaalioloissa kun veren glukoosipitoisuus kasvaa, haima erittää insuliinia, joka auttaa siirtämään glukoosia soluihin ja alentamaan veren glukoosipitoisuutta.
Kun ihminen sairastaa diabetesta, glukoosia ei siirry verestä solujen käyttöön riittävästi sen vuoksi, ettei keho tuota tarpeeksi insuliinia tai insuliinin vaikutus on heikentynyt. Koholla oleva verensokeri voi pitkällä aikavälillä vahingoittaa monia kehon kudoksia, kuten silmiä, munuaisia, sydäntä ja verenkiertoa.

Diabeetikoiden hoidon vuotuiset kokonaiskustannukset ovat yli 11 prosenttia terveydenhuollon kokonaiskustannuksista. Diabeteksen aiheuttamista kustannuksista arviolta 90 prosenttia johtuu lisätautien hoidosta (Stakes)

Ennaltaehkäisy
Kakkostyypin diabeteksen torjumisen keinot ovat painon pitäminen normaalina ja terveellinen ruokavalio. Vihreiden lehtivihannesten syömisen on todettu vähentävän sairastumisriskiä merkittävästi.

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http://fi.wikipedia.org/wiki/Diabetes
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Soursop leaf and bark (Powder), cure diabetes by regulating blood sugar

Last Updated: Saturday, 08 February 2014 02:40
Published: Thursday, 21 February 2013 19:17
Written by Varien Moos

It is a plant that grows in tropical areas in Central America and South America, especially in the Amazon.
It is also known as soursop, Guanábano, Catuche, Catoche, Anona Mexico, Graviola, Anona India, Mole.
The fruit is very delicate dark green covered with soft spines. Is relatively large and very thin shell. Should be harvested before they mature.
The flesh is white, creamy, meaty, juicy and slightly acidic, measuring 20-30 cm long, can weigh 6,8 kg.

All plant parts are used in natural medicine, including bark, leaves, roots and fruits, but the part that contains the greatest concentration of active ingredient is the leaf, where the Annonaceae acetogenins, which have been widely studied from the 1940 that came into use as an insecticide, leading to surprise scientists for its broad power, without causing any harmful effects in animals and man, so they agreed to fund research where, each day discovering new properties, which, as a result of scientific zeal and vested interests, remained in custody for over 20 years. Globalization and parallel studies in Japan and China, they found the light, the wonders of this generous plant.

Soursop leaf and bark (Powder), cure diabetes by regulating blood sugar, which shows its high effectiveness in endocrine commitments: liver, kidney, thyroid, pancreas, ovary, prostate, intestines, muscle relaxant smooth (heart), gall bladder, appendix and fights lung cancer or Lewis, breast cancer and brain tumors, hypotensive, anti-spasmodic, vasodilator, eliminates dust mites that cause asthma and bronchial diseases.

The leaf tea cure liver problems, improves the function of the pancreas. It is effective to deworm children, malaria cure, indicated to raise the defenses in patients with chemotherapy and also for people with HIV (AIDS).
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Various morphological parts of Annona muricata have been reported to be useful as effective remedies against diabetes, hypertension, headache, dizziness, constipation, catarrh, liver problems, neuralgia, rheumatism and arthritis pain (de Almeida, 1993).     http://graviola.fi/tutkimukset-syopa/diabetes/

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Ketoasidoosi


Ketoasidoosi on ketoaineiden liikamäärästä veressä johtuva asidoosi.
Kyseessä on harvinainen ja vaarallinen elimistön poikkeustila. Esimerkiksi paastoamisen tai vähähiilihydraattisen ruokavalion johdosta elimistö siirtyy ketoosiin, jolloin ketoaineiden tuotanto käynnistyy.

Haiman
tuottama insuliini säätelee ketoaineiden tuotantoa, mutta jos haima on vaurioitunut esimerkiksi diabeteksen vuoksi eikä kykene tuottamaan insuliinia, voi seurauksena olla hallitsematon ketoosi, eli ketoasidoosi.
Insuliinin puute aiheuttaa sen, ettei elimistö kykene hyödyntämään veressä olevaa glukoosia vaan alkaa polttaa rasvoja. Se tuottaa myös ketoaineita kudosten energiantarpeeseen (ketoosi). Tästä seuraa elimistön happamoituminen: veren pH on laskenut alle 7,35:n. Asidoosi voidaan todeta verikaasuanalyysillä, ketoosi taas veren tai virtsan ketoainemittauksesta.

Tyypin I diabetekseen sairastuvilla lähes kolmanneksella on ketoasidoosi sairauden toteamishetkellä (lapsilla tyypin II diabetekseen sairastuvilla noin joka kymmenellä), mutta insuliinihoidon alettua tilanne normalisoituu. Myös huonossa hoitotasapainossa oleville diabeetikoille voi kehittyä ketoasidoosi.Tulehdustauti voi heikentää insuliinin tehoa ja näin aiheuttaa ketoosia diabeetikolle.
 - Happomyrkytykseen sairastuneista menehtyy kahdeksan prosenttia.
Ketoasidoosi hoidetaan sairaalahoidossa. Potilaalle voidaan antaa nestehoitoa, ja ketoasidoosi korjautuu yleensä insuliinilla. Ketoasidoosia vaikeammin diagnosoitava ja hoidettava tila on maitohappoasidoosi

Ketoaine on elimistössä rasvasta ja etanolista muodostuva pienimolekyylinen yhdiste.

Ketoaineita muodostuu maksassa ja munuaisissa erityisesti silloin, kun hiilihydraatteja on niukasti saatavilla (kts.ketoosi) ja elimistö joutuu valmistamaan tarvittavan glukoosin itse glukoneogeneesissä.
Elimistössä muodostuu kolmea eri ketoainetta: asetoasetaatti, betahydroksibutyraatti ja asetoni.

Normaalisti ketoaineita syntyy maksan ja munuaisten solujen mitokondrioissa solujen glukoneogeneesin sivutuotteena.
Kun solut tuottavat glukoosia, ne tuottavat tarvitsemansa energian hapettamalla rasvahappoja asetyylikoentsyymi-A:ksi. Asetyylikoentsyymi-A, joka ei hapetu normaalisti sitruunahappokierrossa glukoneogeneesin ollessa käynnissä, muuntuu ketogeneesissä asetoasetaatiksi ja edelleen betahydroksibutyraatiksi.

Ketoaineet kulkeutuvat verenkierron mukana maksasta ja munuisista muualle elimistöön, jossa asetoasetaatti ja betahydroksibutyraatti voidaan käyttää hyväksi. Aivojen gliasolut käyttävät niitä lipidien rakentamiseen. Niitä voidaan hyödyntää myös energiantuotannossa. Erityisesti sydän, lihakset ja aivot käyttävät ketoaineita energianlähteenään.

Asetonia muodostuu spontaanisti asetoasetaatista ja sitä esiintyy paljon vähemmän kuin kahta muuta ketoainetta. Elimistö ei voi hyödyntää asetonia, joten se poistuu kehosta virtsan ja uloshengityksen mukana. Tästä johtuu ketoosissa olevan henkilön hengityksen "makea" tuoksu. Samasta syystä ketoosin voimakkuutta voidaan mitata virtsasta.
More: http://fi.wikipedia.org/wiki/Ketoasidoosi
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DIABETES -TESTI   € 24,90, sis. ALV 14% 

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The Relationship Between Diabetes and Pancreatic Cancer

“It’s complicated.”  But why should pancreatic cancer be any less convoluted than other aspects of the pancreas?  The origin of the embryologic pancreas develops from TWO buds off of the foregut – that EACH “typically” moves and develops, eventually merging to form what we know as the pancreas.  This somewhat individualistic merging process alone produces many anatomic anomalies, as any experienced pancreatic cancer surgeon will tell you.  Also, separately, the function of the pancreas is complicated, divided roughly into exocrine (digestive juice producing) and endocrine (the hormone producing “islet”) areas.
More: http://pancreatica.org/relationship-diabetes-pancreatic-cancer/
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Published online 2013 August 14. doi: 10.3748/wjg.v19.i30.4861.
Is diabetes mellitus a risk factor for pancreatic cancer?  

The relationship between diabetes mellitus and the risk of pancreatic cancer has been a matter of study for a long period of time. The importance of this topic is due to two main causes: the possible use of recent onset diabetes as a marker of the disease and, in particular, as a specific marker of pancreatic cancer, and the selection of a population at risk for pancreatic cancer. Thus, we decided to make an in-depth study of this topic; thus, we carried out an extensive literature search in order to re-assess the current knowledge on this topic.
Even if diabetes is found a decade before the appearance of pancreatic cancer as reported in meta-analytic studies, we cannot select those patients already having non detectable pancreatic cancer, at least with the imaging and biological techniques available today. We believe that more studies are necessary in order to definitively identify diabetes mellitus as a risk factor for pancreatic cancer taking into consideration that approximately 10 years are needed to diagnose symptomatic pancreatic cancer. At present, the answer to the as to whether diabetes and pancreatic cancer comes first similar to the adage of the chicken and the egg is that diabetes is the egg. More: http://www.wjgnet.com/1007-9327/full/v19/i30/4861.htm

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Acidic Diet Increases Risk of Diabetes, Study Says


In an analysis of data from the E3N-EPIC cohort, French women with higher scores on a measure of dietary acidity had about a 70% greater risk of developing diabetes than those whose diets were more alkaline, Guy Fagherazzi, PhD, of Gustave Roussy Institute in Villejuif in France, and colleagues reported online in Diabetologia.

Some work has suggested that Western diets rich in animal products and other acidogenic foods may create an acid load that isn’t compensated for by intake of fruits and vegetables. This can lead to chronic metabolic acidosis, which may play a role in cardiometabolic abnormalities.
Tracking the effects of acidosis

Most importantly from a blood-sugar control perspective, increasing acidosis can reduce the ability of insulin to bind at appropriate receptors in the body, and reduce insulin sensitivity. With this in mind, the scientists decided to analyse whether increased acidosis caused by dietary acid loads increased the risk of type-2 diabetes.
More: http://dailyhealthpost.com/acidic-diet-increases-risk-of-diabetes-study-says/#ixzz3E5IbiOX6
 
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Diabetes and pancreatic cancer: chicken or egg?

Abstract

OBJECTIVES:

Although half of all patients with pancreatic cancer are diabetic at the time of diagnosis, it remains unclear whether the diabetes associated with pancreatic cancer is a cause or an effect of the malignancy.

METHODS:

Epidemiologic studies were reviewed, the geographic prevalence of diabetes and the incidence of pancreatic cancer were examined, and clinical and laboratory studies were reviewed.

RESULTS:

Long-standing diabetes increases the risk of pancreatic cancer by 40% to 100%, and recent-onset diabetes is associated with a 4- to 7-fold increase in risk, such that 1% to 2% of patients with recent-onset diabetes will develop pancreatic cancer within 3 years. Treatment of diabetes or morbid obesity decreases the risk of pancreatic cancer, and metformin therapy decreases the risk due to both its antidiabetic and antineoplastic effects. Recent-onset diabetes associated with pancreatic cancer likely represents secondary or type 3 diabetes. The discrimination of type 3 diabetes from the more prevalent type 2 diabetes may identify the high-risk subgroup of diabetic patients in whom potentially curable pancreatic cancer may be found.

CONCLUSIONS:

Type 2 and type 1 diabetes mellitus increase the risk of pancreatic cancer with a latency period of more than 5 years. Type 3 diabetes mellitus is an effect, and therefore a harbinger, of pancreatic cancer in at least 30% of patients.
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http://www.ncbi.nlm.nih.gov/pubmed/21412116


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Glucagon

     Glucagon has the opposite effect to insulinGlucagon has the opposite effect to insulin
The effects of glucagon are the opposite of the effects induced by insulin. The two hormones need to work in partnership with each other to keep blood glucose levels balanced.

What is glucagon?

Glucagon is a hormone that is produced by alpha cells in a part of the pancreas known as the islets of Langerhans.

The role of glucagon in the body

Glucagon plays an active role in allowing the body to regulate the utilisation of glucose and fats.
Glucagon is released in response to low blood glucose levels and to events whereby the body needs additional glucose, such as in response to vigorous exercise.
When glucagon is released it can perform the following tasks:
  • Stimulating the liver to break down glycogen to be released into the blood as glucose
  • Activating gluconeogenesis, the conversion of amino acids into glucose
  • Breaking down stored fat (triglycerides) into fatty acids for use as fuel by cells

Glucagon and blood glucose levels

Glucagon serves to keep blood glucose levels high enough for the body to function well.
When blood glucose levels are low, glucagon is released and signals the liver to release glucose into the blood.

Glucagon secretion in response to meals varies depending on what we eat:
  • In response to a carbohydrate based meal, glucagon levels in the blood fall to prevent blood glucose rising too high.
  • In response to a high protein meal, glucagon levels in the blood rise.

Glucagon in diabetes

In people with diabetes, glucagon’s presence can raise blood glucose levels too high.
The reason for this is either because not enough insulin is present or, as is the case in type 2 diabetes, the body is less able to respond to insulin.
In type 1 diabetes, high levels of circulating insulin can inhibit the release of glucagon in response to hypoglycemia.
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http://www.diabetes.co.uk/body/glucagon.html

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lauantai 20. syyskuuta 2014

Green tea’s Impact on Cognitive Function is Now Proven!

A new study done at the University of Basil Switzerland,
and led by Stefan Borgwardt, MD, PhD, has now shown that drinking a green tea extract can enhance memory performance. This may have important practical applications for the treatment of neuropsychiatric disorders such as cognitive impairment and Alzheimer’s disease.
Green tea has been discussed for a long time as a tool to use to improve memory. Studies have shown that it does improve cognitive functioning in mice. Several preliminary studies have been done in humans.


This study is unique because it is the “first evidence for the putative beneficial effect of green tea on cognitive functioning, in particular, on working memory processing at the neural system level by suggesting changes in short – term plasticity of the parietal – frontal brain connections”, as stated by the authors.

The special thing about this study is that they used Functional MRI (fMRI), to actually view the subjects brains functioning when they were given green tea in different concentrations or a placebo. The subjects in this study were 12 healthy male students in their early 20’s. The researchers were so careful to avoid any placebo effect, that they did not let the 12 students in the study even drink the green tea drink! They were required to to swallow a feeding tube and given the drink through the tube so that they could not taste it!
Functional MRI scanning allowed the researchers to actually watch the brain working after each of the students “drank” their drink and then did the tasks that they were asked to do while in the fMRI machine. The researchers specifically noticed that there was an increase in connectivity between the parietal lobe of the brain and the frontal lobe of the brain, which went along with the improvement in the performance of tasks that the students were given while they were in the MRI machine.
To me, this is extremely exciting research!
With the new technologies including functional MRI, scientists are now able to prove in very objective ways much of the traditional “folklore” that has been handed down regarding nutritional supplements. This is the gateway to the future of healthcare, where we can individualize treatment for each patient according to their needs.

Certainly, if you have older friends or family who are complaining of memory issues, you can recommend them to have some green tea to help!
Let me know your thoughts!

References
http://www.drsoram.com/greentea/

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Psychopharmacology

© The Author(s) 2014
10.1007/s00213-014-3526-1

Green tea extract enhances parieto-frontal connectivity during working memory processing


(1) Department of Psychiatry (UPK), University of Basel, Wilhelm Klein Str. 27, 4012 Basel, Switzerland
(2) Medical Image Analysis Center, Schanzenstrasse 55, 4031 Basel, Switzerland
(3) Department of Gastroenterology, University Hospital Basel, 4031 Basel, Switzerland
(4) Department of Psychosis Studies, Institute of Psychiatry, King’s College London, London, UK
Stefan Borgwardt 
Received: 7 January 2014 Accepted: 26 February 2014 Published online: 19 March 2014

Abstract
Rationale
It has been proposed that green tea extract may have a beneficial impact on cognitive functioning, suggesting promising clinical implications. However, the neural mechanisms underlying this putative cognitive enhancing effect of green tea extract still remain unknown.

Objectives
This study investigates whether the intake of green tea extract modulates effective brain connectivity during working memory processing and whether connectivity parameters are related to task performance.

Material and methods
Using a double-blind, counterbalanced, within-subject design, 12 healthy volunteers received a milk whey-based soft drink containing 27.5 g of green tea extract or a milk whey-based soft drink without green tea as control substance while undergoing functional magnetic resonance imaging. Working memory effect on effective connectivity between frontal and parietal brain regions was evaluated using dynamic causal modeling.

Results
Green tea extract increased the working memory induced modulation of connectivity from the right superior parietal lobule to the middle frontal gyrus. Notably, the magnitude of green tea induced increase in parieto-frontal connectivity positively correlated with improvement in task performance.

Conclusions
Our findings provide first evidence for the putative beneficial effect of green tea on cognitive functioning, in particular, on working memory processing at the neural system level by suggesting changes in short-term plasticity of parieto-frontal brain connections. Modeling effective connectivity among frontal and parietal brain regions during working memory processing might help to assess the efficacy of green tea for the treatment of cognitive impairments in psychiatric disorders such as dementia.

Image acquisition and analysis
fMRI was performed on a 3T scanner (Siemens Magnetom Verio, Siemens Healthcare, Erlangen, Germany) using an echo planar sequence with a repetition time of 2.5 s, echo time of 28 ms, matrix 76 × 76, 126 volumes and 38 slices with 0.5 mm interslice gap, providing a resolution of 3 × 3 × 3 mm3, and a field of view 228 × 228 mm2. We analyzed fMRI data using SPM8 (http://​www.​fil.​ion.​ucl.​ac.​uk/​spm/​). All volumes were realigned to correct for head movements, mean adjusted by proportional scaling, normalized into standard stereotactic space (Montreal Neurological Institute), and smoothed using a 8 mm full-width at half-maximum Gaussian kernel. We convolved the onset times for each condition (0-back, 1-back, and 2-back) with a canonical haemodynamic response function. Serial correlations were removed using a first-order autoregressive model and a high-pass filter (128 s) was applied to remove low-frequency noise. Six movement parameters were also entered as nuisance covariates to control for movement. We focused our analysis on the 2-back >0-back contrast (main effect of task) to capture the highest possible WM load during the N-back task according to previous N-back fMRI studies (Deserno et al. 2012; Schmidt et al. 2013b).
Differences in local brain activity between the different treatment conditions have previously been reported (Borgwardt et al. 2012); here, we extended this study by conducting an effective connectivity analysis using DCM (Friston et al. 2003), which was restricted to the bilateral superior parietal lobule (SPL) and middle frontal gyrus (MFG).
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http://link.springer.com/article/10.1007/s00213-014-3526-1/fulltext.html
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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4159594/
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http://www.medscape.com/viewarticle/823690


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http://graviola.fi/osta-graviolaa/#!/HIMALAYA-HERBAL-GREEN-TEA-10-bags-India/p/34641112/category=8815702

Green Tea Antioxidants Benefit Skin Health in Women


 
By Greg Arnold, DC, CSCS, July 18, 2014, abstracted from “Green Tea Polyphenols Provide Photoprotection, Increase Microcirculation, and Modulate Skin Properties of Women” in the June 2011 issue of the Journal of Nutrition
When it comes to protecting your skin from the harms of ultraviolet radiation, there is a class of antioxidants called flavonols (1) that maintains skin health and is the reason why consuming cocoa products has been shown to maintain skin health and improve blood circulation (2,3). Beyond cosmetic skin health, flavonols also help protect against skin cancer. This is the most common form of cancer in the United States, with more than 1 million new cases estimated in 2010 (4) and a significant contributor to the $216 total cost of cancer each year (5).
Now a new study (6) suggests that another source of flavonols, green tea, may help maintain skin health in a number of ways. The study involved 60 female subjects between the ages of 40 and 65. For 12 weeks, they consumed either:
-         A green tea beverage providing 1,402 milligrams of a class of flavonols called catechins per day (30 subjects) 
-         A placebo drink (30 subjects)
Before the study began and at weeks 6 and 12, researchers simulated sun exposure to the upper back of each subject, specifically a “minimal erythemal dose (7) of radiation from a solar simulator” and measured skin redness with a method called chromametry (8).
The researchers noted a significant benefit of the green tea supplementation compared to the placebo on skin health. Specifically, skin redness from sun exposure (called UV-induced erythema) was 16% lower than the placebo group by week 6 (p < 0.05) and 25% lower than the placebo group by week 12 (p < 0.05). In addition, the green tea group experienced a 16% decrease in skin roughness, 20% decrease in skin volume, 25% decrease in scaling, and a 17% increase in skin hydration and 20% increase in skin blood flow after 12 weeks compared to the placebo group (p < 0.05).
For the researchers, ““Regular consumption of a beverage rich in tea flavonols contributed photoprotection against harmful UV radiation and helped maintain skin structure and function.”
Greg Arnold is a Chiropractic Physician practicing in Hauppauge, NY.  You can contact Dr. Arnold directly by emailing him at PitchingDoc@msn.com or visiting his web site at www.PitchingDoc.com
Reference:
1.     Dinkova-Kostova AT. Phytochemicals as protectors against ultraviolet radiation: versatility of effects and mechanisms. Planta Med. 2008; 74:1548–59
2.     Heinrich U, Neukam K, Tronnier H, Sies H, Stahl W. Long-term ingestion of high flavanol cocoa provides photoprotection against UVinduced erythema and improves skin condition in women. J Nutr. 2006; 136:1565–9.
3.      Neukam K, Stahl W, Tronnier H, Sies H, Heinrich U. Consumption of flavanol-rich cocoa acutely increases microcirculation in human skin. Eur J Nutr. 2007;46:53–6.
4.     “Skin Cancer” posted on the National Cancer Institute website
5.     “Economic Impact of Cancer” posted on the American Cancer Society Website
6.     Heinrich U. Green Tea Polyphenols Provide Photoprotection, Increase Microcirculation, and Modulate Skin Properties of Women J Nutr 2011 Jun;141(6):1202-8. doi: 10.3945/jn.110.136465
7.     Sies H, Stahl W. Nutritional protection against skin damage from sunlight. Annu Rev Nutr. 2004;24:173–200
8.    
Alaluf S, Heinrich U, Stahl W, Tronnier H, Wiseman S. Dietary carotenoids contribute to normal human skin color and UV photosensitivity. J Nutr. 2002;132:399–403
 
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Vihreä tee on eniten tutkittuja luonnon tuotteita.
Parhaiten tunnetut ainesosat ovat puriinialkaloideja, kofeiini (josta teen yhteydessä joskus käytetään nimitystä teiini), teobromiini, sekä teofylliini, joka on myös tehokas astmalääke.


Katekiinit (tärkein ryhmä)
Ehkäisee ikäihmisten muistin heikkenemistä
Polttaa kaloreita/laihduttaa
Parantaa insuliiniherkkyyttä
Suojaa auringon UV-säteiltä
Estää syöpää
Pienentää kasvainten kokoa
Estää mutaatiota
Estää vapaita radikaaleja
Alentaa kohonnutta kolesterolia
Estää verenpaineen nousua
Tappaa bakteereja
Tappaa influenssaviruksia
Estää kariesbakteereja
Estää pahanhajuista hengitystä


Kofeiini (teiini): Piristää ja virkistää, Diureetti
C-vitamiini:   Ehkäisee stressiä, Ehkäisee flunssaa
B-vitamiinit:    Edistää hiilihydraattien aineenvaihduntaa
Aminobutyyrihappo (GABA): Alentaa kohonnutta verenpainetta
Flavonoidit:     Vahvistaa verisuonten seinämiä
Polysakkaridit:  Alentaa kohonnutta verensokeria
Fluori:      Estää hammasmätää
E-vitamiini:   Hidastaa vanhenemista, Suojaa soluja
Theaiini (aminohappo): Antaa tyypillisen maun vihreälle teelle

Ainesosissa on runsaasti kasviantioksidantteja, jotka tuhoavat elimistössä syntyviä haitallisia vapaita radikaaleja. Niitä syntyy elimistössä ylimäärin sekä ulkoisista että sisäisistä syistä. Ulkoisia aiheuttajia ovat muun muassa ympäristömyrkyt, ilman epäpuhtaudet (kaupunki-liikenne- ja teollisuuspäästöt), otsoni, UV-säteily ja tupakansavu. Sisäisiä syitä radikaalien syntyyn ovat muun muassa tulehdustaudit ja muut pitkäaikaissairaudet (esim. nivelreuma), runsas kahvin ja alkoholin käyttö sekä monet lääkkeet.
Vihreäteeuute nopeuttaa rasvan palamista, todettiin äskettäin tutkimuksessa, joka tehtiin Geneven yliopistossa. Koehenkilöille annettiin vihreää teetä kapseloituna uutteena; puolet koehenkilöistä sai vastaavia lumekapseleita. Tutkijat mittasivat aineenvaihdunnan nopeutta 24 tunnin ajan. Teeuutetta saaneiden kalorit paloivat neljä prosenttia nopeammin ja kokonaisenergian kulutus lisääntyi lähes viisi prosenttia.

Vihreän teen laihduttava vaikutus perustuu sen sisältämiin katekiini-polyfenoleihin.
Tutkijat korostavat nimenomaan, ettei kyse ole pelkästään teen sisältämän kofeiinin (teiinin) vaikutuksesta, vaan kofeiinin ja katekiiniaineiden yhteisvaikutuksesta, joka kiihdyttää aineenvaihduntaa.

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tiistai 16. syyskuuta 2014

8 Year Old Girl Cured of Terminal Stage 4 Cancer with Diet

  

A sad story with a happy ending. And all because of a little colored strip of paper.
Eight-year-old Josie Nuñez was sick for a long time. She felt nauseous and weak and would vomit on a regular basis. She took it in stride like it was normal. Her parents knew it wasn’t normal. They brought her to several doctors but no one could find what was wrong with her.
Finally, one doctor had Josie undergo a CT (computerized tomography) scan to X-ray Josie’s body, looking for anything unusual. They found it.
Josie had a tumor that was pushing against her brain stem. She was very brave when she asked her mother if surgery would hurt and with good nature said “okay” when she was told she’d be asleep during the procedure. After surgery, chemotherapy, and radiation therapy, Josie weighed just 42 pounds.

Seemingly without hope

She seemed to be on the mend but then another tumor was discovered in the middle of her brain; it was therefore inoperable. Doctors told her parents to find a hospice and enjoy the short time they had left with their little girl.
Josie’s parents weren’t willing to give up. They were researching all viable options when they found Dr. Bernardo Majalaca, a San Diego naturopath. Dr. Majalaca asked for Josie’s complete medical history. As is his practice, the very first test he ran was a simple pH test. As he suspected, Josie’s pH level was so acidic, it didn’t register on the litmus strip. His diagnosis: acidosis.
Acidosis is the term given to the condition in which the acid levels in your blood are chronically and dangerously high. “pH” stands for “potential for Hydrogen”: it represents the willingness of a particular substance to share or retain hydrogen atoms. Acids donate hydrogen and bases accept them.
On a scale of 0 to 14, 7 is neutral, 0 is the low acid level and 14 is the high alkaline level. The optimal pH level in our blood is 7.4. When a condition of acidosis occurs, the cells in the body can’t get enough oxygen or nutrition from food and the body basically becomes toxic and cells die (Nobel, Warburg effect).

It is in the nature of cancerous–abnormal–cells to thrive in an oxygen-depleted environment.

In Josie’s case, Dr. Majalaca put her on a strict alkaline diet, prohibiting acidic foods (e.g., meat and fish, bread, dairy, processed foods) and loading up on fresh fruits and vegetables. He prescribed these in juiced form to make them easier for Josie’s body to break down. They consisted of:
Juice 1:
Half a mashed avocado mixed with 8 ounces of freshly juiced carrots. A wooden spoon is used to mash and mix because metal implements can destroy vital enzymes.
Juice 2:
  • 1 Granny Smith apple
  • 1 cup of raw broccoli
  • 1 carrot
  • 5 asparagus spears
  • 1/2 cucumber
  • 1/2 beet
  • 1/2″ slice of pineapple
  • 1/4″ piece of ginger
  • 1/2″piece of Daikon radish
  • A handful of cilantro

What The?!

When Josie went for a check-up with her doctor a few months after beginning this diet, the oncologist was incredulous – she wasn’t expected to have survived that long and, to her amazement, the size of the tumor in Josie’s brain had shrunk seventy-five percent (75%)!  The doctor asked, “what on Earth are you doing?!”
With such fantastic results, Josie continued with Dr. Majalaca’s regimen. At her examination another eight months later, the cancer was gone.
Dr. Majalaca explains, “most doctors have no idea that nutrition has anything to do with cancer”. That’s true: the average medical student receives only 23.9 hours of education in nutrition during the entire medical school career.

A simple test means so much

It is easy to measure your pH level using a litmus test, placing your saliva on a strip of litmus paper and reading the results. If your acid level is consistently below 7, and/or if you experience symptoms of acidosis, there is cause for concern.
You can help to balance the acid and alkaline levels in your body through food. Not enough stress can be put on the importance of pH balance of your body–it affects every cell.
In Josie Nuñez’ case, an insightful doctor had the presence of mind to run a simple test. He knew the implications and how to treat the problem rather than the symptoms. He is the co-author of Josie’s happy ending: a healthy, vibrant young girl with a full life ahead of her.

http://dailyhealthpost.com/eight-year-old-girl-cured-terminal-stage-4-brain-cancer-diet/