SOURSOP IS SAID TO CURE CANCER!!
Keeping Yvonne Kirlew Alive? , I did some research and found some interesting bit
of information that I will share with you.
their money spent on years of research by trying to make a synthetic version of it for
sale. Research shows that with extracts from this miraculous tree it now may be
extreme nausea, weight loss and hair loss
* Protect your immune system and avoid deadly infections
* Feel stronger and healthier throughout the course of the treatment
* Boost your energy and improve your outlook on life
largest drug manufacturers, the fruit of over 20 laboratory tests conducted since the
1970′s! What those tests revealed was nothing short of mind numbing…
Extracts from the tree were shown to:
prostate, lung and pancreatic cancer.
* The tree compounds proved to be up to 10,000 times stronger in slowing the growth
of cancer cells than Adriamycin, a commonly used chemotherapeutic drug!
* What’s more, unlike chemotherapy, the compound extracted from the Soursop tree selectivelyhunts
down and kills only cancer cells. It does not harm healthy cells!
– so why haven’t you heard anything about it? If Soursop extract is as half as promising
as it appears to be–why doesn’t every single oncologist at every major hospital insist on
using it on all his or her patients?
lives(!)–are controlled by money and power.
One of America’s biggest billion-dollar drug makers began a search for a cancer cure
and their research centered on Soursop, a legendary healing tree from the Amazon
fruit-seeds–have been used for centuries by medicine men and native Indians in
South America to treat heart disease, asthma, liver problems and arthritis.
Going on very little documented scientific evidence, the company poured money and
resources into testing the tree’s anti-cancerous properties–and were shocked by the
results. Soursop proved itself to be a cancer-killing dynamo.
so, under federal law, not patentable. There’s no way to make serious profits from it.
synthesize two of the Soursop tree’s most powerful anti-cancer ingredients.
If they could isolate and produce man-made clones of what makes the Soursop so
potent, they’d be able to patent it and make their money back.
Alas, they hit a brick wall. The original simply could not be replicated.
There was no way the company could protect its profits–or even make back the millions
it poured into research.
screeching halt. Even worse, the company shelved the entire project and chose not
to publish the findings of its research!
conscience wouldn’t let him see such atrocity committed. Risking his career, he
contacted a company that’s dedicated to harvesting medical plants from the
Amazon Rainforest and blew the whistle.
When researchers at the Health Sciences Institute were alerted to the news of
Soursop, they began tracking the research done on the cancer-killing tree.
Evidence of the astounding effectiveness of Soursop–and its shocking cover-up–came
in fast and furious….
The results showed that Soursop’s “leaves and stems were found effective in attacking
and destroying malignant cells.” Inexplicably, the results were published in an internal
report and never released to the public…
20 independent laboratory tests, yet no double-blind clinical trials–the typical
benchmark mainstream doctors and journals use to judge a treatment’s value
–were ever initiated…
at Catholic University of South Korea stated that one chemical in Soursop was
found to selectively kill colon cancer cells at “10,000 times the potency of (the
commonly used chemotherapy drug) Adriamycin…”
The most significant part of the Catholic University of South Korea report is that
Soursop was shown to selectively target the cancer cells, leaving healthy cells
Unlike chemotherapy, which indiscriminately targets all actively reproducing cells
(such as stomach and hair cells), causing the often devastating side effects of nausea
and hair loss in cancer patients.
cancer cells among six human cell lines and were especially effective against prostate,
pancreatic and lung cancers.
nopeasti kasvavat, paljon energiaa tuhlaavat syöpäsolut ja estävät niiden ravinnon
ja hapen saannin.
- Graviolan aineosat pysäyttävät syöpäsolujen kasvun jo niiden energian-
tuotannossa, mitokondrioissa, Complex 1: n elektronisiirto -ketjussa.
Electron transport chain COMPLEX 1 (NADH ubiquinone oxidoreductase)
Electron transfer chain in the inner-membrane of mitochondrion. The electron transfer chain contains five complexes designated as complex I, II, III, IV, and V (F 1 F 0 -ATP synthase). The electrochemical H + gradient provided by these membrane-bound complexes serve as energy source for ATP synthesis from ADP and inorganic phosphate by an F 1 F 0 -ATP synthase
postulates that the driver of tumorigenesis is an insufficient cellular respiration caused by insult to mitochondria. The term Warburg effect describes the observation that
cancer cells, and many cells grown in-vitro, exhibit glucose fermentation even when
enough oxygen is present to properly respire. In other words, instead of fully respiring
in the presence of adequate oxygen, cancer cells ferment.
He hypothesized that cancer, malignant growth, and tumor growth are caused by the
fact that tumor cells mainly generate energy (as e.g., adenosine triphosphate / ATP)
by non-oxidative breakdown of glucose (a process called glycolysis).
This is in contrast to "healthy" cells which mainly generate energy from oxidative
breakdown of pyruvate.
Pyruvate is an end-product of glycolysis, and is oxidized within the mitochondria.
(C-32/C34) fatty acids in the polyketide pathway. They are usually characterized by a
combination of fatty acids with a 2-propanol unit at C-2 that forms a methyl-substituted
α,β-unsaturated γ-lactone . Since the discovery of uvaricin from Uvaria accuminata
in 1982, more than 500 AGEs have been identified from different parts of the plants in the
Annonaceae family [73,74]. Due to the special structures and extensive biological activities,
AGEs have attracted significant scientific interest in recent years.
Various biological activities have been reported for AGEs, including antimalarial,
antiparasitic and pesticidal activities [72,75].
However, the biological activities of AGEs are primarily characterized with toxicity
against cancer cells and inhibitory effects against the mitochondrial complex I
(mitochondrial NADH: ubiquinone oxidoreductase) [76,77].
Phytochemical investigations and biological studies on different parts of the A. muricata plant
resulted in the identification of a wide array of AGE compounds, as summarized in Table 1.
The chemical structures of the major acetogenins are shown in Figure 2.
To the best of our knowledge, at the time of preparation (January 2015) of the present
review over 100 AGEs have been identified in A. muricata.
the concentration gradient of the protons "high in the intermembrane space &
low in the matrix of the Mitochondria"
(Scientific Reports volume 6, Article number: 36462 (2016) doi:10.1038/srep36462)
the Annonaceous acetogenins group
- High plasma levels of PAI-1 are related to the development of thrombosis as well as
several other pathologies such as cardiovascular diseases and metabolic disturbances
- Moreover PAI-1 is able to promote tumor angiogenesis and high PAI-1 level in solid
tumors are associated with a poor prognosis 4,5.
In conclusion, our work showed that, as well as their other biological properties,
natural Annonaceous acetogenins, and particularly annonacinone, have an effect on
Indeed, annonacinone is a potent inhibitor of PAI-1 in vitro, ex vivo and in vivo.
Annonacinone mechanism of action and binding site on PAI-1 were also enlightened.
be further studied.
Fibrinolysis is a process that prevents blood clots from growing and becoming
problematic. The fibrinolytic system is closely linked to control of inflammation,
and plays a role in disease states associated with inflammation.Plasmin is produced in
an inactive form, plasminogen, in the liver.
PAI-1 is present in increased levels in various disease states, such as a number of
forms of cancer, as well as in obesity and the metabolic syndrome. It has been linked
to the increased occurrence of thrombosis in patients with these conditions.
The serpin mechanism